Author: Vanessa Ngan, Staff Writer, 2005.
Sunburn is erythema and oedema from excessive exposure to the sun’s rays, more specifically the ultraviolet (UV) radiation emitted from the sun. Sunburn may also occur from exposure to other UV light sources such as solaria or tanning salons.
At a cellular level, sunburn is associated with microscopic changes in the skin. There is the formation of UV induced sunburn cells and a reduction in Langerhans cells and mast cells, which play an essential part of the body's immune defence system.
To better understand the causes of sunburn we need to take a look at some basic principles of the electromagnetic (light) spectrum. This spectrum is divided according to wavelength into the ultraviolet (< 400 nm), visible (400–760 nm), and infrared (> 760 nm). The ultraviolet (UV) spectrum is divided into three broad areas:
UV-C radiation is filtered out or absorbed in the outer atmosphere so does not pose a problem to humans. UV-A and UV-B radiation are the primary causes of sunburn. The skin reacts differently to each waveband.
Reactions to UV-A and UV-B radiation
|Less potent than UV-B but is the wavelength that reaches the surface of the earth most (about 90% at midday)
Penetrates the middle skin layer (dermis) and subcutaneous fat causing damage to the site where new skin cells are created
Long-term exposure causes injury to the dermis resulting in ageing skin
|Much more potent at causing erythema
About 90% is absorbed by the surface skin layer (epidermis)
Epidermis responds by releasing chemicals that cause the reddening and swelling characteristic of the early signs of sunburn
Repeated exposure causes injury to the epidermis resulting in ageing skin
Skin phototyping categorises people into one of six groups based on baseline skin colour and the tendency to tan and burn when exposed to UV radiation.
|Skin Phototype||Typical Features||Tanning ability||MED (mJ/cm2)|
|I||Pale white skin, blue/hazel eyes, blond/red hair||Always burn do not tan||15-30|
|II||Fair skin, blue eyes||Burn easily, tan poorly||25-40|
|III||Darker white skin||Tan after the initial burn||30-50|
|IV||Light brown skin||Burn minimally, tan easily||40-60|
|V||Brown skin||Rarely burn, tan darkly easily||60-90|
|VI||Dark brown or black skin||Never burn always tan darkly||90-150|
People with type I skin phototyping are at much greater risk of sunburn than their type VI counterparts. The amount of UV radiation, measured in energy per unit area, to produce erythema at an exposed site is called the minimal erythema dose (MED), and this is significantly lower in people with a low skin phototype grading.
Other factors that increase the incidence of sunburn include:
The signs and symptoms of sunburn differ according to the skin phototype and length of exposure to UV radiation. Fifteen minutes of midday sun exposure may cause sunburn in a white skin person, while a darker skinned person may tolerate the exposure for hours.
Signs and symptoms usually occur after 2-6 hours of exposure and peak at 12-24 hours; they may include:
In severe cases of sunburn, severe skin burning may result in second-degree burns, dehydration, electrolyte imbalances, secondary infection, shock or even death.
Around 4-7 days after exposure skin may start to peel and flake off.
The treatment of sunburn is to provide relief of the discomfort it can cause with the use of analgesics (pain-killers), cool baths, aloe vera lotions and moisturisers.
However, sunburn is better prevented than treated. Sun protection is your best defence against sunburn and other damaging effects of UV radiation.
An oral food supplement containing Polypodium leucotomas may provide additional oral photoprotection and reduce sunburn.
If you are inadvertently exposed and expect to be sunburned, you may lessen the severity of the burn with the following measures:
It is now clearly apparent that the long-term consequences of overexposure to the sun or other sources of UV radiation are significant. One blistering sunburn at least doubles the likelihood of developing skin cancer later.
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